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However, because tren only causes lean gains, the increased muscle size is more comfortably retained post-cycle. It was discovered that Tren stimulates the activation of the anabolic response protein Akt and results in increased rates of muscle protein synthesis. This suggests that Tren may aid in maintaining an adequate lean mass to ensure that one's gains in strength and hypertrophy are maintained. However, while it is possible for an individual to maximize the effects of training with a moderate intensity of training, most studies have not shown that such an individual can maximize the stimulation of the anabolic response protein Akt. Instead, it has been shown that the stimulation of Akt is increased by training with a lower intensity, as well as with a larger training volume. The main reason for this discrepancy is that it is believed that "hypertrophy", or "building up muscle mass", is a greater concern than muscle hypertrophy, or "storing up muscle mass". This is because the anabolic response protein Akt does not induce an adequate growth stimulus. However, the body's main fuel source, glucose, does provide muscle with energy when it is ingested (gluconeogenesis) so it is plausible that the anabolic response protein Akt does induce an adequate stimulus. Furthermore, it is possible that an anabolic receptor (AR) is involved in Akt stimulation. This would mean that the muscle could be stimulated to produce more protein within the muscle cell. Unfortunately, evidence for the efficacy of such an AR is lacking. Nevertheless, it's difficult to reject the idea that an AR might be present and it would not be impossible to develop a drug that could inhibit AR activity. One way to test this is by administering an anabolic receptor antagonist (ARB) directly to the muscle to decrease the activation of Tren while stimulating the anabolic response protein Akt. However, as with the muscle anabolic response protein, no studies have been conducted on the efficiency of the ARB-mediated stimulation of Tren. It's important to note here that it seems that the anabolic response proteins and Tren synergistically stimulate muscle tissue growth; which means that it's entirely plausible that an ARB could be present and could act as a muscle growth factor and that this could be exploited by the Tren-stimulated anabolic protein Akt. However this would not be possible if neither an ARB nor a Tren existed. Another way to test this may be to use anabolic receptor antagonists that are designed to increase phospholipid content or a substance called choline. The Related Article: